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showed that e-cigarette exposure disrupted lipid homeostasis in alveolar macrophages, leading to the accumulation of lipid-laden macrophages with reduced capacity for pathogen clearance, independent of nicotine content (61)

A notable strength, however, of using both cohorts for this analysis is that they represent different regions of the United States, different socioeconomic backgrounds, and markedly different smoking patterns, with more individuals who currently smoke in the SCCS than in the BWHS

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