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Conclusions: Our study suggested that nicotine activates its downstream signalling to interfere with the ubiquitination process and prevent Bcl-2 from being degraded in lung cancer cells, resulting in the increase of chemoresistance
We then evaluated bone mineral density of femurs divided equally longitudinally serially from distal to proximal points in mice representing two time points: (1) after nicotine had been administered for 3 weeks (nicotine), and (2) 2 weeks after nicotine withdrawal (nicotine cessation) (nicotine, n = 6
Regulation of endogenous gene expression in human non-small cell lung cancer cells by estrogen receptor ligands
As with sales on military bases and Indian reservations, this convenient new way to shop for tax-exempt cigarettes put a major dent in the state's taxed cigarette sales
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