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To this day, there is a prevailing notion that pharmacologically blocking the breakdown of dopamine, serotonin, and other MAO substrates should underlie the antidepressant property of MAO inhibitors by rectifying some neurochemical deficiency, despite the paucity of evidence for any such deficiency syndrome in patients with depression (Quitkin et al., 1979), including the non-selective (MAO-A/B) inhibitors phenelzine (Nardil), isocarboxazid (Thase et al., 1995), and tranylcypromine (Ulrich et al., 2017)

The Cavendish adds a bit if sweet and just a hint if vanilla

To protect the public health of the U.S

Accessed 9/7/2023

Overall rating: 7.5/10 Buy full bottle: Undecided